How do you treat cutis verticis gyrata?

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The treatment of cutis verticis gyrata includes good scalp hygiene to avoid accumulations of secretions in the furrows of the scalp. Definitive treatment by surgery may be requested for cosmetic reasons. Small localised lesions can be excised in one procedure.

How do you reduce CVG?

There is no cure for CVG. Even with applied pressure, the soft and pliant scalp folds cannot be permanently flattened away. Nor are there any medications that will reduce the size or quantity of these soft, spongy wrinkles. The only option to remove the skin folds permanently involves surgery.

Is CVG serious?

Primary essential CVG is seen in otherwise normal individuals with no other abnormalities, and thus any laboratory tests performed during clinical work up are normal, although a low free testosterone level may be present.

Does cutis verticis gyrata affect hair growth?

Skin and Appendages Skin thickening at the vertex causes a peculiar appearance of cutis verticis gyrata (skin folds at the top of the head). Hair growth is increased, and women complain of hirsutism.

How rare is CVG?

Due to the rarity of this disease, the current worldwide prevalence rate is uncertain, but it is estimated that the prevalence in the male population is 1/100,000 and the prevalence in the female population is 0.026/100,000 [2]. CVG is classified into primary essential, primary non-essential and secondary forms [3].

What causes ripples in scalp?

The folds and ridges, that give the appearance of a brain on top of the head, is an indication of an underlying disease: cutis verticis gyrata (CVG). The rare disease causes a thickening of the skin on the top of the head which leads to the curves and folds of the scalp. “There are two forms of it (CVG).

How do you get rid of skin folds in the back of your head?

For example, Rolls of the back of the head that are primarily comprised of fat without much skin excess can be easily treated with some focused liposuction. Rolls of the back of the head that are caused by skin access, in contrast, may require surgical excision of that extra skin.

At what age does CVG occur?

Skin changes may appear in late childhood or during puberty, and usually appear before age 30; hormonal changes have been suggested as a cause of CVG.

Is cutis verticis gyrata a disability?

Primary non-essential cutis verticis gyrata can be associated with neuropsychiatric and ophthalmological abnormalities. This form now has the name cutis verticis gyrata-intellectual disability (CVG-ID).

What causes ripples in your head?

Why is the top of my head wrinkled?

Is it normal to have ridges on your head?

Not everyone has the same skull shape, and normal variations exist among individuals. The skull is not perfectly round or smooth, so it is normal to feel slight bumps and ridges.

What are the treatment options for secondary cutis verticis gyrata?

In mild disease, good scalp hygiene is recommended to avoid accumulations of malodourous secretions in furrows 2. However, in more severe disease, surgical scalp reduction can be considered 2. In patients with secondary cutis verticis gyrata, management should also be focused on the underlying cause 2.

What are the symptoms of cutis verticis gyrata?

Cutis verticis gyrata typically affects the central and back of the scalp, but some forms can involve the entire scalp. The folds are soft and spongy. The folds are unable to be corrected with pressure. The skin colour is not affected. The number of folds can vary from 2 to more than 10.

When was cutis verticis gyrata first described?

Cutis verticis gyrata was first described by Jean-Louis-Marc Alibert (1768-1837), a French dermatologist, in 1837 6. However the term “cutis verticis gyrata” was first coined many years later by Paul Gerson Unna (1850-1929), a German dermatologist, in 1907 6,7.

What causes cutis verticis gyrata (CTG) in Turner syndrome?

Larralde M, Gardner SS, Torrado MV, Fernhoff PM, Santos Munoz AE, Spraker MK, et al. Lymphedema as a postulated cause of cutis verticis gyrata in Turner syndrome. Pediatr Dermatol. 1998 Jan-Feb. 15 (1):18-22.